Preliminary experiments indicate conolidine may perhaps inhibit precise ion channels, cutting down neuronal excitability and restricting pain indicators. This system is especially pertinent in neuropathic soreness, where abnormal signaling results in persistent pain. In addition, conolidine seems to impact G protein-coupled receptor (GPCR) pathways

Preliminary experiments point out conolidine may perhaps inhibit certain ion channels, lessening neuronal excitability and limiting pain signals. This mechanism is particularly relevant in neuropathic discomfort, wherever abnormal signaling will cause persistent distress. Additionally, conolidine seems to influence G protein-coupled receptor (GPCR)